This can lead to a range of responses including: cell death, survival, differentiation, proliferation, and migration.2 TNFα cannot usually be detected in healthy individuals; however, elevated serum and tissue levels are found in inflammatory and infectious conditions, and serum levels correlate to the severity of infections.2 Lack of regulation of TNFα production has been implicated in a variety diseases such as Alzheimer’s disease, cancer, and Crohn’s disease.2-4
This Sword Assay has been optimized for use with the R&D Systems DuoSet ELISA for Human TNFα (DY210 or DY210-05).
Table 2. TNFα levels were quantified in human plasma EDTA from healthy donors using the R&D Systems Human TNFα DuoSet ELISA (DY210) with Sword Assay for Human TNFα. Donor samples were tested in duplicate in three separate runs.
Table 3. Human TNFα Reference Standard was spiked into pooled human serum from healthy donors. Human TNFα levels were quantified using the R&D Systems Human TNFα DuoSet ELISA (DY210) with Sword Assay for Human TNFα.
Table 4. Human TNFα Reference Standard was spiked into pooled human plasma EDTA from healthy donors. Human TNFα levels were quantified using the R&D Systems Human TNFα DuoSet ELISA (DY210) with Sword Assay for Human TNFα.
Table 5. Human TNFα was quantified in human serum from twelve healthy donors using the R&D Systems Human TNFα DuoSet ELISA (DY210) with Sword Assay for Human TNFα.
Table 6. Human TNFα was quantified in human plasma EDTA from twelve healthy donors using the R&D Systems Human TNFα DuoSet ELISA (DY210) with Sword Assay for Human TNFα.
Citations
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